Biology of intracranial aneurysms: role of inflammation
Intracranial aneurysms (IAs) linger as a potentially devastating clinical problem. Despite intense investigation, our understanding of the mechanisms leading to aneurysm development, progression and rupture remain incompletely defined.
Cerebral Aneurysm Sac Growth as the Etiology of Recurrence after Successful Coil Embolization
Coil compaction is thought to be the main mechanism for recurrence in cerebral aneurysms with previously successful coil embolization. We hypothesize that sac growth may be equally or more important.
Management and Surgical Treatment of Ruptured Intracranial Aneurysms
PMC1935149: The advantages, methods and results of surgical intracranial obliteration of aneurysms in conjunction with the use of intra-cranial or neck ligation of arteries were studied in 177 patients made up of the following groups.
The Lancet : The defi nition of stroke has undergone a transformation in recent years,4 and the importance of control of cardiovascular risk factors to reduce stroke mortality and the value of inclusion of stroke in cardiovascular risk
Stromal Cell-Derived Factor-1 Associated with Angiogenesis and Inflammatory Cell Infiltration
PMC3877706: Ruptured cerebral aneurysms are characterized by inflammation-mediated wall remodeling. We studied the role of stromal cell-derived factor-1 (SDF-1) in inflammation-mediated wall remodeling in cerebral aneurysms.
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